<font size="4" style="color:rgb(0, 51, 102);"><br/> <font size="3">以我們熟悉的網路查詢,輸入中文或英文關鍵字,得到的結果是:<br/> 約有66,400項符合"退化性膝關節炎"的查詢結果<br/> 更有251,000項符合"Osteoarthritis of Knee"的查詢結果<br/> 歸納這些有關退化性膝關節炎的資訊,提供的觀念不外乎:<br/> <br/> *確切病因仍屬不明<br/> *目前的各種治療方法只能減輕症狀,無法阻止退化<br/> *軟骨組織一旦受損就無法再生<br/> *最終將走上置換人工關節一途</font> <br/> <br/> <font size="3">也就是說:<br/> <br/> 若想發展任何有關退化性膝關節炎的創新觀念,<br/> <br/> 必將面臨哥白尼式的挑戰!</font><br/> <br/> </font><font size="4" style="color:rgb(0, 51, 102);"><font size="3"><font size="4" style="color:rgb(0, 51, 102);"><font size="3"></font></font></font></font>
保險公司以文中最後三項,剔除我新加保的醫療險中可能因
退化性膝關節炎引起的相關給付,包括人工關節置換,甚至
將來老了,因雙膝行走不便從樓上跌下來引發的骨科治療。
申辯加抗議,以呂主任的研究為例,保險公司說,無此說法
及支持文獻。
無奈~~消極抵制,不加保,不讓保險公司賺錢。
期待~~
踏腳石重新擺放的那一天,可以很豪邁的把資料,丟給保險
公司,但,還是不加保。
非常了解這種感覺!!
4.哥白尼式的挑戰,退化性膝關節炎的創新觀念 Here is a report about
the human body generates new blood vessels and nerves trying to
heal the damage. The removal of fiber like plica tissue and
cleaning inside of medial compartment provide very friendly
enviroment to help the healing of the damaged cartilage.
Quote from the article : (This report may support your healing
of 退化性膝關節炎的創新觀念 is possible.)
We speculate that a chemotactic response to products of disc
breakdown is responsible for the proliferation of vascularity
and CGRP-containing sensory nerves found in the endplate region
and vertebral body adjacent to degenerate discs. The
neuropeptides substance P and CGRP have potent vasodilatory as
well as pain-transmitting effects. The increase in sensory nerve
endings suggests increase in blood flow, perhaps as an attempt
to augment the nutrition of the degenerate disc. ..
(Max 1000 Ch.)
Thank you for your information and supportive statement. I do wish more researchers would join in this important field.
Thanks for your quick response. Recently I was trying to
understand why OA causing pain. There is barely nerve ending
,blood vessel in the cartilage, disc except at the endplate or
at one third of Meniscus pad, if they are worn out then no
sensory nerve left just like "nerve block" surgery
then should
clear the pain but why still have pain which bothered me. The
following article explaining the proliferation of blood vessels
and accompanying nerve fibres in the degenerated disc, bone, may
provide nutrition path to make your KHPO works.
http://lib.bioinfo.pl/meid:130312
In US the "removal of medial plica" is not popular neither
recognized as potential source of Knee OA. You applied "finite
element model" to analyze the knee OA is very impressive. You
are the pioneer 哥白尼 to theorize "pieces of the cartilage are
chipped away from the bones into the wei
ght-bearing part of the
joint, it causes irregular and excessive rubbing".
After totally reset my knowledge about OA knee according to the concept of KHPO, I surprisingly find that I could solve >95% of knee problem in my daily practice. Of course, the chief complaint of these patients is always pain! Although the origin of pain in OA knee is still controversial, in my point of view, more and more recent studies support my theory of MAS. I have very positive expectation about the future of KHPO and hope researchers in this field could appreciate the simple concept of MAS.